Autoimmune hepatitis
● Symptomata et causas
Causam: HLA et non-HLA moleculis tum environmental triggers ut virus, toxins, et microbiome sunt suggesserant ut key components in T cellula-mediata immune responsio.
Cellular et Menismular Menismi: De Presentation de autoantigenic peptide (AG) ad Navis CD4 + T adiutorium cellulis (Th0) by DENTRIGING (VI-Dendritiones (VI-Dends, TH1, et TH1, Th17 cellulis. TH1 cellulis secare II-II et ifn-y, quod stimulate CD8 + cellulis inducere expressio HLA classis ego et HLA class II moleculis in Hepatocystes. Trogs et Th2 cellulis secreto ii, IV, II-X, et II-XIII ita stimulating in c cellulis et plasma cellulis quod se producere autoantibodiis. Th17 cellulis, qui incrementum numerum per gradus iecoris fibrosis, secreto proinflammatory cytokines et supprimere T regulatory cellulis (TreG). De numero decrescit de Triggs ducit ad imminendum tolerantia ad autoantigens, quae deinde results in initiation et perpetuatione autoimmune iecur damnum.
Autoimmune hepatitis
● Symptomata et causas
Causam: HLA et non-HLA moleculis tum environmental triggers ut virus, toxins, et microbiome sunt suggesserant ut key components in T cellula-mediata immune responsio.
Cellular et Menismular Menismi: De Presentation de autoantigenic peptide (AG) ad Navis CD4 + T adiutorium cellulis (Th0) by DENTRIGING (VI-Dendritiones (VI-Dends, TH1, et TH1, Th17 cellulis. TH1 cellulis secare II-II et ifn-y, quod stimulate CD8 + cellulis inducere expressio HLA classis ego et HLA class II moleculis in Hepatocystes. Trogs et Th2 cellulis secreto ii, IV, II-X, et II-XIII ita stimulating in c cellulis et plasma cellulis quod se producere autoantibodiis. Th17 cellulis, qui incrementum numerum per gradus iecoris fibrosis, secreto proinflammatory cytokines et supprimere T regulatory cellulis (TreG). De numero decrescit de Triggs ducit ad imminendum tolerantia ad autoantigens, quae deinde results in initiation et perpetuatione autoimmune iecur damnum.