Acuti respiratorii angustia syndrome (ARDS)
● Symptomata et causas
Figura IV: Systemic versus alveolaris inflammatio in progressionem acuti respiratorii angustia syndrome
Systemica et alveolaris inflammatio non necessario correlated in aegris cum acuti respiratorii angustia syndrome (ARDS). The panels show differences between systemic hypoinflammatory (A, C) and hyperinflammatory (B, D) inflammation, and the differences between alveolar hypoinflammatory (A, B) and hyperinflammatory (C, D) inflammation. Etsi haec tabulata illustrare extrema condiciones systemica sine alveolaris inflammatio et alveolaris sine systemica inflammatione, severitatem systemica et alveolaris inflammatio existit in spectro quod probabiliter variat aliquanto a patientes estote, conferunt ad heterogeneity. (A) in normalis Alveolus, sine inflammatione vel injuriam. (B) mutationes in Hyperinflamatory subpheothpe, quod est propria systemica inflammatio, endothelial distemperantia, et coagulation. Sine alveolaris inflammatio, injuriam causatur inflammatio agitatam a systemica cellula ad alveolar cellula (flavo sagitta) unde auctus permeabilland et alveolar Oedema. (C) mutationes in aegris cum alveolaris hyperinflamation sine systemica Hyperinflamatory subpheothpe. Alveolaris cellulis Epithelial, Alveolaris macrophages et neutrophils habere centralis munus in proinflammatory cytokine productio. Citithelial cellulis et macrophages sunt essentialis in productione proinflammatory moleculis. Neutrophils producere variis iniuria moleculis, quod damnum typus I et type II pneumocytes inde in auctus campester of pneumocyte iniuriam venalicium. Sine systemica inflammatio, injuriam per inflammationem in hoc missione repulsi ab alveolaris ad systemica cellula (flavo sagitta), etiam inde in auce et alveolar OEDEMA. (D) et combined coram systemica et alveolaris hyperinflamation. Sub his inflammatio inducit pulmonem injuriam, auctus permeability et alveolar OEDEMA.
Doi: 10.1016 / S0140-6736 (XXII) 01485-4
Acuti respiratorii angustia syndrome (ARDS)
● Symptomata et causas
Figura IV: Systemic versus alveolaris inflammatio in progressionem acuti respiratorii angustia syndrome
Systemica et alveolaris inflammatio non necessario correlated in aegris cum acuti respiratorii angustia syndrome (ARDS). The panels show differences between systemic hypoinflammatory (A, C) and hyperinflammatory (B, D) inflammation, and the differences between alveolar hypoinflammatory (A, B) and hyperinflammatory (C, D) inflammation. Etsi haec tabulata illustrare extrema condiciones systemica sine alveolaris inflammatio et alveolaris sine systemica inflammatione, severitatem systemica et alveolaris inflammatio existit in spectro quod probabiliter variat aliquanto a patientes estote, conferunt ad heterogeneity. (A) in normalis Alveolus, sine inflammatione vel injuriam. (B) mutationes in Hyperinflamatory subpheothpe, quod est propria systemica inflammatio, endothelial distemperantia, et coagulation. Sine alveolaris inflammatio, injuriam causatur inflammatio agitatam a systemica cellula ad alveolar cellula (flavo sagitta) unde auctus permeabilland et alveolar Oedema. (C) mutationes in aegris cum alveolaris hyperinflamation sine systemica Hyperinflamatory subpheothpe. Alveolaris cellulis Epithelial, Alveolaris macrophages et neutrophils habere centralis munus in proinflammatory cytokine productio. Citithelial cellulis et macrophages sunt essentialis in productione proinflammatory moleculis. Neutrophils producere variis iniuria moleculis, quod damnum typus I et type II pneumocytes inde in auctus campester of pneumocyte iniuriam venalicium. Sine systemica inflammatio, injuriam per inflammationem in hoc missione repulsi ab alveolaris ad systemica cellula (flavo sagitta), etiam inde in auce et alveolar OEDEMA. (D) et combined coram systemica et alveolaris hyperinflamation. Sub his inflammatio inducit pulmonem injuriam, auctus permeability et alveolar OEDEMA.
Doi: 10.1016 / S0140-6736 (XXII) 01485-4