Eosinophilic esophagitis (Eoe)
● Symptomata et causas
Early-Vita Exposures, geneticae factores, et in atopic re publica verisimile augmentum morbo susceptibilitatem in eisinophilic Esophagitis. Patefacio ut antigens causet in Esophageal Epithelium dimittere metrinas, II-XXXIII, et organorum stromal lymphopoietin (tslp). Haec Cytokines rursus stimulate T-adiutorem type II (Th2) cellulis, secretionem II-XIII, II, IV et II-V. II-XIII et II-IV stimulate mutationes videatur in Esophageal Epithelii, comprehendo basalis cell hyperplasia et dilatatur intracellular spatia. Chemotaxins, ETAXIN-III et II-V, ducunt ad Granulocyte infiltration. Et mixta Cytokine Milieu etiam confert ad activation of Fibrblasts in Linea propria, collagen depositione, et TEXTUS rigorem.
Doi: 10.1001 / Jama.201.14920.
Eosinophilic esophagitis (Eoe)
● Symptomata et causas
Early-Vita Exposures, geneticae factores, et in atopic re publica verisimile augmentum morbo susceptibilitatem in eisinophilic Esophagitis. Patefacio ut antigens causet in Esophageal Epithelium dimittere metrinas, II-XXXIII, et organorum stromal lymphopoietin (tslp). Haec Cytokines rursus stimulate T-adiutorem type II (Th2) cellulis, secretionem II-XIII, II, IV et II-V. II-XIII et II-IV stimulate mutationes videatur in Esophageal Epithelii, comprehendo basalis cell hyperplasia et dilatatur intracellular spatia. Chemotaxins, ETAXIN-III et II-V, ducunt ad Granulocyte infiltration. Et mixta Cytokine Milieu etiam confert ad activation of Fibrblasts in Linea propria, collagen depositione, et TEXTUS rigorem.
Doi: 10.1001 / Jama.201.14920.